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The ability of IL-1 to induce chondrocyte catabolism [121] has been well known for a long time, but it is only recently that IL-1 has been shown to disrupt chondrocyte iron homeostasis [117] and trigger chondrocyte ferroptosis [22, 122]

Unlike the other three components that primarily target structural repair and regeneration, KPV addresses inflammatory signaling at the molecular level

Glutathione is a small peptide molecule produced by every cell of

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